Suppression of HIV-1 replication by microRNA effectors
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* Corresponding authors: Yamina Bennasser yamina.bennasser@igh.cnrs.fr - Monsef Benkirane monsef.benkirane@igh.cnrs.fr
- † Equal contributors
Retrovirology 2009, 6:26 doi:10.1186/1742-4690-6-26
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BioMed Central: 4 citations
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Research
Kenneth W Witwer, Andria K Watson, Joel N Blankson, Janice E Clements Retrovirology 2012, 9:5 (12 January 2012) miRNA profiles, obtained using multiple acquisition, data processing, and analysis methods, distinguished ES and uninfected controls from viremic HIV-1-infected patients. For several miRNAs, however, ES and viremic patients shared similar expression patterns. Differentially expressed miRNAs included those with reported roles in HIV-1 latency (miR-29 family members, miRs -125b and -150). Others, such as miR-31 and miR-31*, had no previously reported connection with HIV-1 infection but were found here to differ significantly with uncontrolled HIV-1 replication.
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Research
Zachary Klase, Laurent Houzet, Kuan-Teh Jeang Cell & Bioscience 2011, 1:38 (23 November 2011) |
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Review
Laurence Colin, Carine Van Lint Retrovirology 2009, 6:111 (4 December 2009) |
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Minireview
Human cellular restriction factors that target HIV-1 replication Klaus Strebel, Jeremy Luban, Kuan-Teh Jeang BMC Medicine 2009, 7:48 (16 September 2009) Although different host cell factors act to inhibit viral replication and limit infection, HIV-1 has a number of measures to counteract this function; Strebel, Luban and Jeang review our current understanding of these interactions.
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